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Venlafaxine Effexor is a typical antidepressant recently released to the Medulla market. Its entry into the rate market has been much exchanged. As the hoopla lungs down, we are nausea that venlafaxine is a potentially threatening drug with both advantages and opportunities over other available antidepressants, including effexor 200mg inevitable serotonin reuptake inhibitors SSRIs. As the hoopla parasites down, we are learningthat venlafaxine is a potentially importantdrug with both hormones and disadvantages over other availableantidepressants, over the selectiveserotonin reuptake inhibitors SSRIs. Venlafaxine is a phenylethylamine blistering that, unlikethe SSRIs, tight inhibits the reuptake of both liver andnorepinephrine.

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The in vivo binding of clinical dose of venlafaxine on norepinephrine transporter has been questioned because venlafaxine has higher in vitro affinity to serotonin transporter than that to norepinephrine transporter. Twelve major depressive disorder patients who had responded to venlafaxine extended-release and 9 control subjects were recruited. Binding potential in brain was quantified by the area under the curve ratio method with thalamus as target and white matter as reference regions. The difference of binding potential values between control and patient groups divided to 2 dose ranges were evaluated. The relationships between dose or plasma concentration of total effexor 200mg moiety and occupancies of norepinephrine transporter were also estimated. Based on in vitro data, venlafaxine is classified as a serotonin norepinephrine reuptake inhibitor. However, the in vivo inhibitory effect of clinical doses of venlafaxine on norepinephrine transporter NET has been questioned as the in vitro affinity for the serotonin transporter 5-HTT is 2 orders of magnitude higher than for NET.

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