If results of other anti-PFB treatments are unsatisfactory, surgical intervention with laser therapy provides a viable option that could result in a cure. PFB can occur in any ethnic group; however, individuals with tightly coiled hair e. Some women may develop PFB, especially those with hirsutism or those who routinely shave their bikini area. The pathogenesis of PFB is thought to be multifactorial. However, transfollicular penetration and extrafollicular penetration have been identified as the two primary processes responsible for the initial development of the PFB lesion FIGURE 1.

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Naturally occurring glucocorticoids hydrocortisone and cortisone, which also have salt retaining properties, are used in replacement therapy in adrenocortical deficiency states. Their synthetic analogs are used primarily for their anti-inflammatory effects in disorders of many organ systems. Hematologic Disorders: Acquired autoimmune hemolytic anemia, congenital erythroid hypoplastic anemia Diamond Blackfan anemia, pure red cell aplasia, select cases of secondary thrombocytopenia. Miscellaneous: Trichinosis with neurologic or myocardial involvement, tuberculous meningitis with subarachnoid block or impending block when used concurrently with appropriate antituberculous chemotherapy. Neoplastic Diseases: For palliative management of: leukemias and lymphomas. Nervous System: Cerebral edema associated with primary or metastatic brain tumor or craniotomy. Renal Diseases: To induce diuresis or remission of proteinuria in idiopathic nephrotic syndrome, or that due to lupus erythematosus. Rheumatic Disorders: As adjunctive therapy for short-term administration to tide the patient over an acute episode or exacerbation in acute gouty arthritis; acute rheumatic carditis; ankylosing spondylitis; psoriatic arthritis; rheumatoid arthritis, including juvenile rheumatoid arthritis selected cases may require low-dose maintenance therapy. For the treatment of dermatomyositis, polymyositis, and systemic lupus erythematosus. DEPO-MEDROL is indicated as adjunctive therapy for short-term administration to tide the patient over an acute episode or exacerbation in acute gouty arthritis, acute and subacute bursitis, acute nonspecific tenosynovitis, epicondylitis, rheumatoid arthritis, synovitis of osteoarthritis.

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It was originally discovered in A governmentally-recognized ID which uniquely identifies the product within its regulatory market. Erythromycin is indicated in the treatment of infections caused by susceptible strains of various bacteria. Mild to moderate upper respiratory tract infections caused by Streptococcus pyogenes, Streptococcus pneumoniae, or Haemophilus influenzae when used concomitantly with appropriate doses of sulfonamides can be treated with erythromycin. It is effective in eliminating the causative organism from the nasopharynx of infected individuals, rendering them noninfectious.

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Prominent symptoms among patients with diabetic gastroparesis include early satiety, nausea, and vomiting. Patients often have associated end organ damage such as peripheral neuropathy, nephropathy, or retinopathy. Gastric emptying mechanisms are deranged to include vagal nerve neuropathy, reduction in intrinsic inhibitory neurons critical for motor conduction, a reduction in gastric pacemaker cells interstitial cells of Cajal, and increased glucagon levels. Neurohormonal dysfunction and hyperglycemia also reduce the frequency of antral contractions. Behavioral, medical, and surgical treatment strategies of gastroparesis are outlined in Table Treatment for diabetic gastroparesis involves measures to enhance gastric motility and emptying.

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